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When botulinum toxin is injected near a motor end plate, where a trigger point would develop, this reduces the motor nerves ability to drive a sustained taut band in the muscle, which has been previously reported as part of an autism-like movement disorder in children. "Although most motor movements are automatic and are not necessarily pathological, they may be compromised by the way we are born and how we treat our movements when we are very young," said Michael Voll, PhD, director of MIT's Salk Institute for Biological Studies and lead author of the study. "We hope this research could lead to new avenues for therapies to treat the problem. "Our current results, while exciting, represent the first detailed evidence that this mechanism is a plausible underlying mechanism for motor symptoms of autism in young children. More research is needed to confirm or refute our findings. In addition, we need confirmation from future small clinical trial." The paper, "Autism-like motor deficits in young mouse with taut band observed in motor, sensory and brainstem regions," was published July 14, 2014, in the Journal of Cerebral Blood Flow & Metabolism. In addition to Voll and co-first authors Andrew B. Hsu, PhD, and Joshua A. Oleson, PhD, of the Salk Institute, Dana R. Toth, PhD, professor of psychiatry and neuroscience and senior author on the paper, is a professor of biomedical engineering and head of the Department of Electrical and Computer Engineering from MIT. Autism Spectrum Disorder is a neurodevelopmental disorder characterized by impairments in social communication, communication with parents and other caregivers, and repetitive behavior in children. The symptoms most commonly associated with autism are language delay, repetitive behavior, and restricted interests such as dolls, food, and repetitive behaviors. Related Article:
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